Debbie Potts Coaching

First of all… What is LEAKY GUT?

What is Leaky Gut Syndrome?

Leaky gut syndrome (also known as increased intestinal permeability) is a term used by experienced leaky gut doctors in Tampa to describe the condition of the intestines when its lining becomes damaged and gaps appear between the cells.

These gaps allow large particles and toxins to pass through the intestinal wall and escape into the body. Because these “foreign objects” are not supposed to be in the blood they trigger the immune system, which sets up inflammation in different parts of the body and a cascade of symptoms such as headaches, fatigue, brain fog, food sensitivities, bloating, cramps, and more.

Leaky gut is not widely recognized by mainstream physicians but as Dr. Oz recently mentioned, it is often overlooked because conventional medicine doesn’t know how to diagnose it, and a patient can present with multiple, seemingly unrelated symptoms.

Because a leaky gut is responsible for so many unexplained chronic health conditions, it is one of the first things we test for and address. Repairing a leaky gut is one of the most important foundational steps to recover from any chronic disease.

Leaky Gut Symptoms

Leaky gut often causes uncomfortable pains, sensations, feelings, and symptoms in other parts of the body. Patients can feel fatigued, have brain fog, feel depressed, have unexplained pains or even an overactive immune system.

Common leaky gut symptoms include:

Causes of Leaky Gut

Many factors are implicated such as:

  • nutritional deficiencies
  • poor diet
  • eating irritating foods
  • food sensitivities
  • amino acid deficiencies
  • chronic stress
  • environmental toxins
  • pesticides
  • chemicals & additives
  • GMO foods
  • inflammation
  • autoimmunity
  • systemic toxicity
  • yeast
  • fungi
  • mold
  • bacterial overgrowth
  • antibiotics and certain medications

Natural Leaky Gut Treatment

If you are found to have a leaky gut, our top gut doctor will prescribe a natural leaky gut treatment program which will be developed to address the underlying reason(s) why you developed it.

Depending on the underlying causes and your current situation, treatment may include one or more of the following:

Part of your natural leaky gut treatment will include resolving any secondary damage caused by having a leaky gut.

Don’t Put off Addressing Gut Dysfunction

Getting your gut health on-point and healing any outstanding issues should not be put on the back burner. Gut health issues, like leaky gut, affect all areas of your health. Your gut is the foundation for the strength and proper function of your immune system. In addition it plays a vital role in nutrient absorption, waste elimination, energy production, and brain health.

Gut issues that are left untreated tend to develop into more serious issues, like autoimmune conditions, Crohn’s, IBS, arthritis, joint pains, neurological disorders, and even cardiovascular disease.

What can seem like a minor gut annoyance, if left untreated, over time can snowball into a more serious condition. Don’t let that be you. Get it checked and fix it while it’s easy and cost-effective to do so.


What is LPS?

LPS stands for Lipopolysaccharide. It’s a molecule found in the outer membrane of Gram-negative bacteria, which are a type of bacteria characterized by the structure of their cell walls. LPS is considered an endotoxin because it’s released when these bacteria die and their cell walls break down.

Dr. Tom O’Bryan talks about Leaky Gut & LPS…

Excessive Lipopolysaccharides (LPS). LPS is a type of endotoxin produced inside your body.

LPS is like a poison. Small amounts in your bloodstream trigger the production of an anti-inflammatory hormone, adiponectin.

That’s great!

But LPS is the perfect example of too much of a good thing.

Too much actually creates inflammation.

If it stays in the gut, it isn’t a huge issue.

But with leaky gut, it triggers inflammation in the body when it enters the bloodstream

What are gram negative bacteria?

  • Gram-negative bacteria are a group of bacteria that have a distinct cell wall structure, which includes an outer membrane composed of lipopolysaccharides.
  • This outer membrane acts as a protective barrier for the bacteria and helps them to survive in various environments.
  • LPS molecules consist of a lipid portion (lipid A), a core polysaccharide, and an O-specific polysaccharide chain.
  • LPS plays a crucial role in the structural integrity of Gram-negative bacteria and also serves as an endotoxin when released into the environment or bloodstream, triggering immune responses in animals and humans.

Gram-negative bacteria are a diverse group of bacteria that possess a characteristic cell wall structure that stains pink or red in the Gram staining method.

Some common examples of Gram-negative bacteria include:

  1. Escherichia coli (E. coli): Found in the intestines of humans and animals, some strains can cause gastrointestinal infections.
  2. Salmonella spp.: Pathogenic strains can cause foodborne illnesses such as salmonellosis.
  3. Pseudomonas aeruginosa: An opportunistic pathogen that can cause infections in wounds, burns, and the respiratory system, particularly in immunocompromised individuals.
  4. Klebsiella pneumoniae: Known for causing pneumonia and urinary tract infections, especially in hospital settings.
  5. Neisseria gonorrhoeae: The causative agent of gonorrhea, a sexually transmitted infection.
  6. Neisseria meningitidis: Can cause meningitis and septicemia.
  7. Vibrio cholerae: The bacterium responsible for cholera, a diarrheal disease transmitted through contaminated water and food.
  8. Helicobacter pylori: Associated with gastritis, peptic ulcers, and an increased risk of stomach cancer.
  9. Bordetella pertussis: Causes whooping cough (pertussis), a highly contagious respiratory disease.
  10. Haemophilus influenzae: Can cause respiratory tract infections, including pneumonia and meningitis, as well as other invasive diseases.

These are just a few examples, and there are many more Gram-negative bacteria with diverse characteristics and roles in infectious diseases and environmental processes.

What are endotoxins?

Endotoxins are toxins that are an integral part of the bacterial cell and are released when the cell disintegrates.

Endotoxemia refers to the presence of endotoxins, particularly LPS, in the bloodstream. When endotoxins enter the bloodstream, they can trigger an immune response leading to inflammation and potentially serious health consequences. Endotoxemia can result from infections caused by Gram-negative bacteria or from conditions where bacteria normally residing in the gut enter the bloodstream due to intestinal permeability issues (leaky gut syndrome).

The effects of endotoxemia on blood glucose, insulin, and triglycerides can vary depending on the individual and the severity of the endotoxemia. In general, endotoxemia can induce metabolic disturbances. Some studies suggest that acute exposure to endotoxins can lead to insulin resistance, which may result in elevated blood glucose and insulin levels. Additionally, endotoxemia has been associated with dyslipidemia, including elevated triglyceride levels. However, the specific effects may depend on factors such as the duration and severity of endotoxemia, as well as individual metabolic characteristics.

What are signs and symptoms of endotoxemia?

Endotoxemia occurs when endotoxins, such as lipopolysaccharides (LPS) from Gram-negative bacteria, enter the bloodstream and trigger an immune response. The signs and symptoms of endotoxemia can vary depending on the severity of the condition and individual factors. Some common signs and symptoms may include:

  1. Fever: Endotoxemia often causes an increase in body temperature as part of the immune response.
  2. Chills and rigors: Patients may experience sudden onset chills or rigors, which are involuntary muscle contractions often associated with fever.
  3. Hypotension: Endotoxemia can lead to a drop in blood pressure, potentially causing dizziness, lightheadedness, or fainting.
  4. Tachycardia: Increased heart rate is a common response to endotoxemia, as the body attempts to compensate for decreased blood pressure.
  5. Tachypnea: Rapid breathing may occur as the body tries to increase oxygen delivery to tissues.
  6. Malaise and weakness: Patients may feel generally unwell, fatigued, or weak.
  7. Gastrointestinal symptoms: Endotoxemia can cause nausea, vomiting, abdominal pain, and diarrhea.
  8. Headache: Some individuals may experience headaches as a result of endotoxemia.
  9. Altered mental status: In severe cases, endotoxemia can lead to confusion, delirium, or loss of consciousness.
  10. Systemic inflammatory response syndrome (SIRS): Endotoxemia may trigger a systemic inflammatory response characterized by abnormalities in body temperature, heart rate, respiratory rate, and white blood cell count.

It’s important to note that endotoxemia can progress to more severe conditions such as sepsis, septic shock, and multiple organ dysfunction syndrome (MODS) if not promptly treated. If someone experiences symptoms suggestive of endotoxemia, particularly if they have a known infection or risk factors for Gram-negative bacterial infections, they should seek medical attention immediately for proper evaluation and management.

Dr. Tom O’Bryan on STRESS, LEAKY GUT + LPS

  1. The stress hormones weaken and damage your gut lining, leading to leaky gut.
  2. Excess stress will trigger leaky gut all by itself.
  3. LPS breach the cell walls and floods out into your bloodstream.
  4. Your body senses more stress triggers more inflammation.
  5. You begin a never-ending cycle.
  6. You have an on-off switch in your body.
  7. You are either in “fight or flight” mode, or “rest and digest” mode.
  8. The body is designed to stay in the “rest and digest” mode where it functions best.
  9. Unfortunately, more often than not, you are in “fight or flight” mode.
  10. Not only is your body not in its relaxed reparation state, but it also starts to shut down systems.
  11. Then it enters emergency mode.
  12. The trick here is offering your body as much support as possible.
  13. This limits stress and allows it to get back to its proper restorative state.

Lipopolysaccharides (LPS) are large molecules found in the outer membrane of Gram-negative bacteria, which are commonly associated with infections and inflammatory responses in the gut.

While LPS itself is not directly related to small intestinal bacterial overgrowth (SIBO) or small intestinal fungal overgrowth (SIFO), there are indirect connections between them:

  1. Intestinal Permeability: Both SIBO and SIFO can contribute to increased intestinal permeability, also known as leaky gut syndrome. In conditions of SIBO or SIFO, overgrowth of bacteria or fungi in the small intestine can lead to disruption of the intestinal barrier, allowing substances such as LPS to pass into the bloodstream more easily.
  2. Inflammatory Response: Overgrowth of bacteria or fungi in the small intestine can trigger an inflammatory response in the gut mucosa. This inflammation can further compromise the integrity of the intestinal barrier, potentially leading to increased translocation of LPS and other microbial components into the bloodstream.
  3. Systemic Effects: Elevated levels of LPS in the bloodstream, resulting from increased intestinal permeability and inflammation associated with SIBO or SIFO, can contribute to systemic inflammation and immune activation. This systemic inflammation may exacerbate symptoms and contribute to the progression of gastrointestinal disorders.
  4. Immune Dysregulation: Chronic exposure to LPS and other microbial products due to SIBO or SIFO can lead to immune dysregulation, with implications for both local gut health and systemic immune function. Dysregulated immune responses may further perpetuate inflammation and disrupt the balance of gut microbiota.

While the relationship between LPS and SIBO/SIFO is complex and multifactorial, addressing intestinal dysbiosis and restoring gut barrier integrity are essential components of managing these conditions.

Strategies to address SIBO and SIFO may include dietary modifications, antimicrobial therapy, probiotics, and gut barrier support.

By addressing the underlying dysbiosis and intestinal permeability, it may be possible to reduce systemic inflammation and mitigate the impact of LPS on overall health.

Here are some functional lab markers that may provide clues to theseLeaky Gut conditions:

  1. LPS and Endotoxins:
    • LPS Binding Protein (LBP): LBP is a marker that can indicate the presence of circulating LPS in the bloodstream. Elevated levels of LBP suggest increased exposure to LPS and potential endotoxemia.
    • Endotoxin Core Antibodies (EndoCAb): EndoCAb levels may reflect the immune response to LPS exposure. Lower levels of EndoCAb can indicate higher levels of circulating endotoxins.
  2. SIBO:
    • Hydrogen and Methane Breath Test: This test measures the levels of hydrogen and methane gas produced by bacteria in the small intestine after the ingestion of a substrate (e.g., lactulose or glucose). Elevated levels of hydrogen and/or methane can indicate bacterial overgrowth in the small intestine.
    • Small Intestinal Culture: Culturing a sample obtained via endoscopy can directly identify bacterial overgrowth in the small intestine.
    • Small Bowel Aspirate: Aspirating fluid from the small intestine during endoscopy can be analyzed for bacterial overgrowth using various methods, including quantitative culture or PCR-based techniques.
  3. SIFO:
    • Fungal Culture: Culture of a small intestinal aspirate or stool sample can identify the presence of fungal overgrowth in the small intestine.
    • Fecal Calprotectin: Elevated levels of calprotectin in the stool can indicate intestinal inflammation, which may be associated with fungal overgrowth or other gastrointestinal conditions.
    • Organic Acid Test (OAT): OAT can detect metabolites produced by fungi and bacteria in the gastrointestinal tract, providing indirect evidence of fungal overgrowth.

Additionally, comprehensive stool analysis tests (CSA) can offer insights into dysbiosis, inflammation, and other markers associated with gastrointestinal health. These tests often include assessments of microbial diversity, markers of inflammation, and levels of short-chain fatty acids (SCFAs), which can provide additional information relevant to LPS, endotoxins, SIBO, and SIFO.

It’s important to interpret these laboratory tests in the context of the patient’s clinical presentation and other diagnostic findings to guide appropriate treatment decisions. Working with a healthcare provider experienced in interpreting functional laboratory tests can help ensure accurate assessment and effective management of gastrointestinal issues.

Elevated levels of glucose, insulin, A1C (glycated hemoglobin), and triglycerides are often associated with metabolic dysfunction and conditions such as insulin resistance, type 2 diabetes, and dyslipidemia. While lipopolysaccharides (LPS) and endotoxins are primarily associated with immune responses and inflammation, there is evidence to suggest that they may also play a role in metabolic disturbances and the development of metabolic disorders.

Here’s how LPS and endotoxins may be related to these metabolic markers:

  1. Insulin Resistance: Chronic exposure to LPS and endotoxins has been linked to the development of insulin resistance, a condition where cells become less responsive to the effects of insulin. LPS-induced inflammation can impair insulin signaling pathways, leading to decreased glucose uptake by cells and elevated blood glucose levels. Insulin resistance is a key component of metabolic syndrome and type 2 diabetes.
  2. Glucose Dysregulation: LPS-induced inflammation may directly affect pancreatic beta cells, which are responsible for producing insulin. Inflammatory cytokines released in response to LPS exposure can impair beta cell function and insulin secretion, contributing to elevated blood glucose levels and impaired glucose tolerance.
  3. A1C: Hemoglobin A1C (HbA1c) reflects average blood glucose levels over the past 2-3 months and is used as a marker of long-term glycemic control. Chronic inflammation associated with elevated LPS and endotoxins may contribute to sustained hyperglycemia, leading to higher levels of glycated hemoglobin (HbA1c) over time.
  4. Triglycerides: LPS-induced inflammation can also affect lipid metabolism and contribute to dyslipidemia, characterized by elevated triglyceride levels. Inflammatory cytokines can stimulate hepatic production of triglycerides and reduce clearance of triglyceride-rich lipoproteins from the bloodstream, leading to elevated circulating triglycerides.

Overall, chronic exposure to LPS and endotoxins can contribute to metabolic dysfunction through multiple mechanisms, including inflammation-induced insulin resistance, impaired glucose regulation, and dyslipidemia.

  • Addressing gut health, reducing inflammation, and improving metabolic function may help mitigate the impact of LPS and endotoxins on metabolic markers and reduce the risk of developing metabolic disorders.
  • Lipopolysaccharide (LPS)-induced inflammation refers to the inflammatory response triggered by the presence of LPS, a component of the outer membrane of Gram-negative bacteria, in the body.
  • When LPS enters the bloodstream, typically due to increased intestinal permeability (leaky gut) or bacterial infections, it can activate the immune system and lead to the production of pro-inflammatory cytokines and other inflammatory mediators.
  • This inflammatory response plays a key role in the body’s defense against bacterial infections but can also contribute to various health problems when dysregulated.

The mechanism of LPS-induced inflammation involves several steps:

  1. Recognition by Immune Cells: LPS binds to Toll-like receptor 4 (TLR4), which is primarily expressed on immune cells such as macrophages and dendritic cells. Binding of LPS to TLR4 triggers a signaling cascade that activates these immune cells.
  2. Activation of NF-κB: Activation of TLR4 by LPS leads to the activation of nuclear factor kappa B (NF-κB), a transcription factor that regulates the expression of pro-inflammatory genes. NF-κB activation results in the production of pro-inflammatory cytokines such as tumor necrosis factor-alpha (TNF-α), interleukin-1 beta (IL-1β), and interleukin-6 (IL-6).
  3. Release of Pro-inflammatory Mediators: Pro-inflammatory cytokines produced in response to LPS, along with other inflammatory mediators such as prostaglandins and reactive oxygen species (ROS), contribute to the inflammatory response. These molecules recruit immune cells to the site of infection or injury and promote inflammation.
  4. Amplification of Inflammation: LPS-induced inflammation can lead to a positive feedback loop, where the production of pro-inflammatory cytokines further activates immune cells and amplifies the inflammatory response.

When we have leaky gut and high LPS levels, several consequences can occur:

  1. Systemic Inflammation: Increased intestinal permeability allows LPS to pass from the gut lumen into the bloodstream, leading to systemic inflammation. Chronic exposure to high levels of LPS can contribute to low-grade inflammation throughout the body, which is associated with various chronic diseases, including metabolic syndrome, cardiovascular disease, and autoimmune disorders.
  2. Immune Dysregulation: Chronic exposure to LPS can dysregulate the immune system, leading to aberrant immune responses and autoimmune reactions. The persistent activation of immune cells by LPS can contribute to tissue damage and autoimmune conditions.
  3. Metabolic Dysfunction: LPS-induced inflammation and immune activation can disrupt metabolic homeostasis, leading to insulin resistance, dyslipidemia, and impaired glucose regulation. These metabolic disturbances contribute to the development of metabolic syndrome, type 2 diabetes, and other metabolic disorders.
  4. Impact on Gut Health: High levels of LPS and chronic inflammation can further compromise gut barrier function, perpetuating leaky gut syndrome and creating a vicious cycle of inflammation and gut dysfunction.

Overall, leaky gut and high LPS levels contribute to systemic inflammation, immune dysregulation, metabolic dysfunction, and gut health issues, which can have profound implications for overall health and increase the risk of developing various chronic diseases.

Addressing gut health, reducing inflammation, and restoring gut barrier integrity are important strategies for mitigating the effects of leaky gut and high LPS levels on health.

What are some Nutritional Therapy Solutions??  

Sure, here’s a breakdown of the foods and benefits associated with polyphenols, prebiotics, fermented foods, digestive enzymes with HCL betaine, berberine, NAC (N-acetylcysteine), omega fatty acids, green tea, and curcumin:

  1. Polyphenols:
    • Foods: Berries (such as blueberries, strawberries, raspberries), dark chocolate, red grapes, apples, citrus fruits, cherries, pomegranates, green tea, coffee, nuts (especially almonds and walnuts), seeds, olives, extra virgin olive oil, herbs (such as parsley, thyme, rosemary), and spices (such as cinnamon, cloves, turmeric).
    • Benefits: Polyphenols have antioxidant and anti-inflammatory properties, and they may help protect against chronic diseases such as heart disease, cancer, diabetes, and neurodegenerative disorders. They also support gut health by promoting the growth of beneficial gut bacteria and reducing inflammation in the gut.
  2. Prebiotics:
    • Foods: Garlic, onions, leeks, asparagus, artichokes, bananas, apples, chicory root, Jerusalem artichokes, dandelion greens, flaxseeds, and seaweed.
    • Benefits: Prebiotics are non-digestible fibers that serve as food for beneficial gut bacteria. They help promote the growth and activity of these bacteria, which in turn support digestion, immune function, and overall gut health.
  3. Fermented Foods:
    • Foods: Yogurt, kefir, sauerkraut, kimchi, miso, tempeh, kombucha, pickles, natto, and traditionally fermented soy products.
    • Benefits: Fermented foods contain beneficial probiotic bacteria that support gut health by replenishing and diversifying the microbiota, improving digestion, enhancing nutrient absorption, and modulating the immune system.
  4. Digestive Enzymes with HCL Betaine:
    • Benefits: Digestive enzymes with HCL betaine can support digestion by helping break down macronutrients (proteins, carbohydrates, fats) and improve nutrient absorption. HCL (hydrochloric acid) betaine helps maintain an acidic environment in the stomach, which is necessary for optimal digestion and nutrient absorption.
  5. Berberine:
    • Benefits: Berberine is a compound found in several plants, including goldenseal, barberry, and Oregon grape. It has been shown to have anti-inflammatory, antimicrobial, and blood sugar-lowering effects. Berberine may help support gut health by modulating the gut microbiota and reducing inflammation in the gut.
  6. NAC (N-acetylcysteine):
    • Benefits: NAC is a precursor to glutathione, a powerful antioxidant that plays a key role in detoxification and immune function. NAC supplementation may support gut health by reducing oxidative stress, promoting mucosal healing, and supporting liver detoxification.
  7. Omega Fatty Acids:
    • Foods: Fatty fish (such as salmon, mackerel, sardines, trout), flaxseeds, chia seeds, hemp seeds, walnuts, and algae.
    • Benefits: Omega-3 fatty acids (found primarily in fatty fish and certain plant sources) have anti-inflammatory properties and support cardiovascular health, brain function, and immune function. Omega-3s also help maintain the integrity of cell membranes, including those in the gut.
  8. Green Tea:
    • Benefits: Green tea contains polyphenols, particularly catechins, which have antioxidant and anti-inflammatory properties. Green tea consumption has been associated with numerous health benefits, including improved heart health, weight management, and reduced risk of certain cancers.
  9. Curcumin (from Turmeric):
    • Benefits: Curcumin is the active compound in turmeric, a spice widely used in traditional medicine. Curcumin has potent anti-inflammatory and antioxidant properties and may help support gut health by reducing inflammation, modulating the gut microbiota, and promoting mucosal healing.

Incorporating a variety of these foods and supplements into your diet can help support gut health, reduce inflammation, and promote overall well-being. However, it’s important to consult with a healthcare professional before starting any new supplements, especially if you have underlying health conditions or are taking medications.

Do you need a biofilm disruptor?

Biofilm disruptors are substances or compounds that interfere with the formation, stability, or structure of biofilms. Biofilms are complex communities of microorganisms (such as bacteria, fungi, and algae) that adhere to surfaces and are encased in a matrix of extracellular polymeric substances (EPS). These biofilms can form on various surfaces, including medical implants, catheters, dental plaque, and natural environments like rocks and water pipes.

Biofilms play a significant role in many aspects of human health and environmental processes. They can contribute to chronic infections, impair wound healing, cause dental plaque formation, contaminate water systems, and degrade industrial equipment. Biofilms also provide protection to microorganisms against antimicrobial agents and host immune responses, making them challenging to eradicate.

Biofilm disruptors aim to target and dismantle biofilms, making microorganisms more susceptible to antimicrobial treatments and host defenses. They can work by various mechanisms, including:

  1. Interfering with Quorum Sensing: Quorum sensing is a communication system used by bacteria within biofilms to coordinate their activities and regulate biofilm formation. Biofilm disruptors may interfere with quorum sensing signals, preventing biofilm development.
  2. Breaking Down Extracellular Matrix: The extracellular matrix (EPS) of biofilms provides structural support and protection to microorganisms within the biofilm. Biofilm disruptors may contain enzymes or agents that degrade or disrupt the EPS, destabilizing the biofilm structure.
  3. Inhibiting Adhesion: Some biofilm disruptors prevent the initial attachment of microorganisms to surfaces, reducing biofilm formation. These agents may interfere with the expression of adhesion molecules or create an unfavorable environment for microbial attachment.
  4. Disrupting Cell-to-Cell Communication: Biofilm disruptors may target the signaling pathways involved in cell-to-cell communication within biofilms, inhibiting microbial cooperation and biofilm development.

Examples of biofilm disruptors include enzymes (such as DNase and dispersin B), antimicrobial peptides, chelating agents (such as ethylenediaminetetraacetic acid, EDTA), surfactants, quorum sensing inhibitors, and natural compounds derived from plants or microorganisms.

Research into biofilm disruptors is ongoing, with the aim of developing new strategies to combat biofilm-related infections, improve medical device safety, and address environmental contamination issues. However, it’s essential to consider the potential impacts of biofilm disruptors on microbial ecology and to ensure their safety and efficacy before widespread use in clinical or environmental settings.

What does Dr. Davis share in his book SUPERGUT?

“Super Gut” is a book by Dr. William Davis that focuses on improving gut health and overall well-being through a comprehensive four-week program. While I don’t have access to the specific details of the program outlined in the book, Dr. Davis is known for advocating a grain-free and low-carbohydrate approach to diet and lifestyle. Here are some general principles that may be included in the “Super Gut” program:

  1. Elimination of Grains: Dr. Davis emphasizes the elimination of grains, particularly wheat and gluten-containing grains, from the diet. This is based on the premise that grains, especially modern hybridized varieties, can contribute to gut inflammation, dysbiosis, and autoimmune conditions in susceptible individuals.
  2. Low-Carbohydrate Diet: The program may promote a low-carbohydrate, high-fat diet (often referred to as a ketogenic or keto-friendly diet) to help stabilize blood sugar levels, reduce inflammation, and support gut health. This typically involves consuming plenty of non-starchy vegetables, healthy fats, moderate amounts of protein, and minimal processed carbohydrates and sugars.
  3. Emphasis on Gut Healing Foods: The program may include recommendations for gut-healing foods such as bone broth, fermented vegetables, and foods rich in prebiotic fibers. These foods can help support the growth of beneficial gut bacteria, repair intestinal epithelial cells, and improve gut barrier function.
  4. Probiotics and Supplements: Dr. Davis may recommend specific probiotic supplements and other supplements known to support gut health, such as digestive enzymes, glutamine, and certain vitamins and minerals.
  5. Stress Reduction and Lifestyle Factors: The program may also address lifestyle factors that impact gut health, such as stress reduction techniques, adequate sleep, regular physical activity, and minimizing exposure to environmental toxins.
  6. Gradual Reintroduction and Personalization: After the initial elimination phase, the program may involve a gradual reintroduction of certain foods to assess individual tolerance and identify triggers for gut symptoms. This approach allows for personalization of the diet based on individual needs and sensitivities.

Super Gut shows readers how to eliminate bad bacteria and bring back the missing “good” bacteria with a four-week plan to reprogram your microbiome based on research and techniques that not only get to the root of many diseases but improve levels of oxytocin (the bonding/happy hormone), brain health, and promote anti-aging, weight loss, mental clarity, and more restful sleep. Super Gut explains the science clearly and includes more than 40 recipes, a diet plan, and resources so you can pinpoint your gut issues, correct them, and rebuild long-term health and well-being.Just as modern humans have introduced dramatic disruptions of the external world environment, so we have also disrupted our internal ecosystems, the composition of the microbes dwelling in our gastrointestinal (GI) tracts. Not only have we lost species that used to provide us with important functions, we have also allowed unhealthy species to proliferate and occupy parts of the GI system where they do not belong. This process underlies an astounding number of health problems from obesity, to Type 2 diabetes, to skin rashes, to neurodegenerative conditions, to many forms of cancer.

These insights into the disrupted human microbiome mean that everything we know about human diseases needs to be reconsidered. The good news: Many lost microbes can be restored with often spectacular health results and unhealthy bacteria that have come to occupy the entire GI tract can be pushed back resulting in relief from numerous health conditions. And this can all be accomplished from the comfort of your own home.

Modern people have lost important microbes from their natural microbiomes
Restoring lost microbes can yield astounding health benefits including smoother skin with reduced wrinkles, deeper sleep, reduced appetite, acceleration of healing, increased empathy and other effects
1 in 3 people have allowed unhealthy pathogenic microbes to take over the entire length of the gastrointestinal tract, a process that can be experienced as irritable bowel syndrome, fibromyalgia, or restless leg syndrome, and also lead to skin rashes such as rosacea, autoimmune conditions, depression, neurodegenerative conditions such as Parkinson’s disease, even colon cancer.
Identify, then manage, this overgrowth of unhealthy microbes from the comfort of your own home without prescription medication
Restore lost microbes using a unique method of making yogurt that yields extraordinarily high counts of bacteria with resultant amplified health benefits

The Supergut Probiotic Yogurt 4-Week Plan


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